SAE Media Group stories: The 14th Annual RNA Therapeutics Conference is set to return in February 2023 with a key concentrate on mRNA therapeutic purposes.
LONDON, UK, October 20, 2022 /EINPresswire.com/ — SAE Media Group is delighted to announce the 14th Annual RNA Therapeutics Conference, going down on 8 – 9 February 2023, in London, UK. The 2023 Conference theme is on investigating the Next Generation of Genetic Medicine Through RNA Based Therapies.
Chair for the convention is business knowledgeable Heinrich Haas, Vice President RNA Formulation and Drug Delivery, BioNTech.
Interested events can register for the convention at: http://www.therapeutics-rna.com/PR1ein and benefit from the early chicken provide to save lots of £400 which expires on 31 October 2022.
By attending the convention attendees may have the chance to
• Explore how mRNA is main the best way within the genetic medication area as the subsequent era of novel drug modalities
• Investigate the newest traits in AI drug discovery as a instrument for designing novel RNA primarily based therapeutics
• Enhance your data of the newest improvements in RNA software and RNA supply strategies with case research from BioNTech, Neuway Pharma and Ethris GmbH
• Engage within the rising advances and improvement of novel RNA molecules within the remedy of persistent respiratory, neurodegenerative, and infectious ailments
Building on the success of earlier years of the RNA Portfolio, the 14th Annual RNA Therapeutics Conference will convey collectively business consultants from large pharma and main biotechs to achieve an knowledgeable and holistic view on the newest developments of the business.
View the agenda and speaker line – up at http://www.therapeutics-rna.com/PR1ein
For sponsorship enquiries contact Andrew Gibbons at [email protected] or +44 (0) 20 7827 6156
For media enquiries contact Marketing, Lamprini Balasi at
[email protected] or +44 (0) 20 7827 6732
14th Annual RNA Therapeutics
8 – 9 February 2023
London, UK
#RNATx2023
http://www.therapeutics-rna.com/PR1ein
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